Are Covid-19 vaccines promoting the formation of stealthily adapted coronavirus variants?

Public health officials seem unaware of the likely role of Covid-19 vaccines in accelerating the development of stealthily adapted coronaviruses. In fact, they have not yet accepted the existence of widespread human infections with viruses derived from stealthily adapted monkeys. These viruses were inadvertently introduced into humans from polio vaccines. This occurred as a consequence of the use of polio vaccines grown in kidney cells grown from monkeys infected with cytomegalovirus.

An erroneous assumption is that current covid-19 vaccines provide immunity comparable to natural infections. This is clearly not so. First, the vaccine is administered by intramuscular injections, while natural infections occur through the respiratory mucosa. Intramuscular injections are not particularly effective in stimulating the development of mucosal immunoglobulin A (IgA) antibodies or resident cytotoxic T lymphocytes (CTLs). The reduced level of vaccine-induced mucosal immunity means that following exposure to the SARS-CoV-2 virus, a proportion of vaccinated persons will likely acquire a persistent subclinical infection that is limited to the superficial respiratory mucosa. Public Health authorities allude to this possibility by insisting that those who are immunized must continue to wear masks. However, persistent low-level infections will provide an opportunity for virus variants to emerge. Some of these will be more infectious, while others will be better able to evade the immunity triggered by the vaccine and thus spread further throughout the body.

The second big difference between the vaccine and natural infection is that the FDA allows the use of only one component of the virus in the vaccine, namely the spike protein. It is much easier to modify a virus, or even eliminate it, from a single component than to make simultaneous changes to the multiple antigens targeted by immunity against natural infections. Removal of the spike protein is possible since coronaviruses have other means of entering cells. The virus can then more easily undergo changes in the remaining genes that encode the relatively few components of the virus that are normally targeted by cellular immunity.

The persistence of subclinical infections due to the relative lack of mucosal immunity achieved by intramuscular injections and the systemic immune response restricted to only the spike protein, may lead more rapidly than natural infection to the formation of stealthily adapted coronaviruses. A corollary to this premise is that the English, South African, and Brazilian variants likely originated from individual participants in the Covid-19 vaccine trials conducted in each of the countries. With wider use of the vaccine, many more variants are expected, including stealthily adapted coronaviruses.

The stealthy adaptation has another very worrying characteristic. It is the incorporation of additional genetic sequences that is likely required for the virus to regain infectivity. The added sequences can come from the cellular genome and from the genomes of other microbes. This has allowed, for example, stealthily adapted viruses derived from the polio vaccine to deliver monkey cell sequences to humans.

The brain is particularly susceptible to symptomatic diseases caused by viruses adapted to stealth. These viruses can be cultured from patients with chronic fatigue syndrome (CFS) and also from children with autism. Long Covid syndrome has many clinical features in common with CFS. Until proven otherwise, Long Covid Syndrome should be considered a viral illness with the potential for person-to-person transmission, including during pregnancy. It is critical to start culturing blood samples from Long Covid syndrome patients and sequencing the resulting viruses.

Although the cellular immune system will not normally engage with stealthily adapted viruses, they can still be suppressed via the alternative cellular energy (ACE) pathway. It is likely that this pathway preceded photosynthesis in plants and the production of energy by all forms of life from the metabolism of food. In humans and animals, the brain is probably the main receptor of vital energy for the ACE pathway. The attracted energy is then transferred to the body fluids where it is expressed as additional kinetic activity. The energy is called KELEA, an abbreviation for Kinetic Energy Limiting Electrostatic Attraction. KELEA can also be added to water, which is then called KELEA excellent water. Portable bags containing this water and inhalation of mists from the water are being evaluated as simple means of improving the ACE pathway. These approaches can apparently suppress both conventional and stealthy viral diseases.

A more detailed discussion of these issues can be found in the book “Stealth Adapted Viruses; Alternative Cellular Energy (ACE) & KELEA Activatedf Water”. Support information is also available at the following sites:

https://zenodo.org/record/4489960#.YBizzS2cbEY

https://zenodo.org/record/4489960#.YBmpCC1h1N1

Leave a Reply

Your email address will not be published. Required fields are marked *